African Swine Fever by W. R. Hess (auth.), Yechiel Becker (eds.)

By W. R. Hess (auth.), Yechiel Becker (eds.)

African swine fever (ASF) is attributable to an outbreak that's labeled as a member of the Iridovirinae relatives. The illness within the warthog, the typical host, in Africa was once defined in 1921 by way of R. E. Montgomery. The reservoir of the vi rus is inti cks. The i ntroduct i on of household pi gs into territory occupied via warthogs i nf ected wi th ASF within the 1960's has endangered the pig around the globe. The family pig is extremely delicate to ASF and develops a devastating affliction that kills the pig with out giving the immune procedure an opportunity to protect the animal opposed to the virus an infection. the facility of ASF virus to contaminate and spoil cells of the reticuloendothelial procedure leaves a defenseless host that succumbs to infection that could be defined as an obtained immune deficiency di sease of family pi gs. creation of the virus into Iberia within the 1960's resulted in a sequence of ASF epidemics in Spain and Portugal . . and later in France, that brought on heavy financial losses. among 1976 and 1960, ASF virus made its visual appeal in Malta and Sardinia . . in addition to in Brazil, The Dominican Republic . . Haiti, and later in Cuba. In 1985-6 . . ASF seemed in Belgium and The Netherlands.

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Polatnick, J. R. Arch. Ges. Virusforsch. 38: 383-385. 32. , Ortin, J. and Vinuela,E. J. Gen. Virol. 93: 253-258, 1978. 33. Ortrn, J. and Vlnuela, E. J. Virol. 21, 902-905, 1977. 34. M. G. Virology 33: 173-175, 1967. 35. Tabares, E. and Sanchez, Botija, C. Arch. Virol. 61: 4959, 1979. 36. , Shimizu, M. R. Am. J. Vet. Res. 41: 1357-1367. 37. W. L. Cell 27: 391-401, 1981. 38. , Venkatesan, S. and Moss, B. Cold Spring Harbor Symp. Quant. BioI. 47: 723-729, 1983. 39. , Enjuanes, L. andiVinuela, E. J.

Copyright © 1987. Martinus Nijhoff Publishmg. Boston, All rights reserved. 52 with this virus may recover and resist challenge with the same strain of ASFV, but the mechanisms of recovery and resistance have not yet been defined (6-8); therefore, a satisfactory vaccine has not yet been developed either. Further knowledge of viral proteins can contribute to studies as to their role in the immunology and control of the disease. With regard to the latter, the major structural and infectious protein (VP73) is being used as a diagnostic reagent (9) in epidemiological studies.

5. R. In: Virology Monographs, vol. 9 (Eds. S. Gard, C. F. Meyer), Springer, Beling Heidelber, New York, pp 1-33, 197!. 6. O. and Coggins, L. Am J. Vet. Res. 30: 1343-1359, 1969. 7. J. Arch. ges. Virus Forsch 20: 164-179, 1967. 8. A. and May, D. Am. J. -Res. ~: 104-108, 1960. 9. , Enjuanes, L. and Vifiuela, E. J. Virol. 52:3746, 1985. 10. J. C. Br. Vet. J. 134: 280282, 1978. 11. F. L. Cornrn. Eur. Cornrn. EUR 8466 EN, pp. 132-142, 1983. 12. , Cubero, I. and Vifiuela, E. J. Gen. Virol. 34: 455-463, 1977.

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